Histone Acetyltransferase-Dependent Pathways Mediate Upregulation of NADPH Oxidase 5 in Human Macrophages under Inflammatory Conditions: A Potential Mechanism of Reactive Oxygen Species Overproduction in Atherosclerosis

Lignende værker

• Pharmacological inhibition of histone deacetylase reduces NADPH oxidase expression, oxidative stress and the progression of atherosclerotic lesions in hypercholesterolemic apolipoprotein E-deficient mice; potential implications for human atherosclerosis
  af: Simona-Adriana Manea, et al.
  Udgivet: (2020-01-01)
• Pharmacological inhibition of histone deacetylase reduces NADPH oxidase expression, oxidative stress and the progression of atherosclerotic lesions in hypercholesterolemic apolipoprotein E-deficient mice; potential implications for human atherosclerosis
  af: Manea, Simona-Adriana, et al.
  Udgivet: (2019)
• Epigenetic regulation of vascular NADPH oxidase expression and reactive oxygen species production by histone deacetylase-dependent mechanisms in experimental diabetes
  af: Manea, Simona-Adriana, et al.
  Udgivet: (2018)
• Detection of Vascular Reactive Oxygen Species in Experimental Atherosclerosis by High-Resolution Near-Infrared Fluorescence Imaging Using VCAM-1-Targeted Liposomes Entrapping a Fluorogenic Redox-Sensitive Probe
  af: Manea, Simona-Adriana, et al.
  Udgivet: (2021)
• C/EBP transcription factors regulate NADPH oxidase in human aortic smooth muscle cells
  af: Manea, Simona-Adriana, et al.
  Udgivet: (2014)