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Activation of BNIP3-mediated mitophagy protects against renal ischemia–reperfusion injury
Acute kidney injury (AKI) is a syndrome of abrupt loss of renal functions. The underlying pathological mechanisms of AKI remain largely unknown. BCL2-interacting protein 3 (BNIP3) has dual functions of regulating cell death and mitophagy, but its pathophysiological role in AKI remains unclear. Here,...
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| Foilsithe in: | Cell Death Dis |
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| Main Authors: | , , , , , , , , , , , |
| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
Nature Publishing Group UK
2019
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6742651/ https://ncbi.nlm.nih.gov/pubmed/31515472 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41419-019-1899-0 |
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