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Soluble β-Amyloid(1-40) Induces NMDA-Dependent Degradation of Postsynaptic Density-95 at Glutamatergic Synapses
Amyloid-β (Aβ) has been implicated in memory loss and disruption of synaptic plasticity observed in early-stage Alzheimer's disease. Recently, it has been shown that soluble Aβ oligomers target synapses in cultured rat hippocampal neurons, suggesting a direct role of Aβ in the regulation of syn...
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| 出版年: | J Neurosci |
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| 主要な著者: | , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Society for Neuroscience
2005
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6725651/ https://ncbi.nlm.nih.gov/pubmed/16319306 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.3034-05.2005 |
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