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Armet, a UPR-upregulated protein, inhibits cell proliferation and ER stress-induced cell death
The accumulation of misfolded proteins in the endoplasmic reticulum (ER) causes ER stress that initiates the unfolded protein response (UPR). UPR activates both adaptive and apoptotic pathways, which contribute differently to disease pathogenesis. To further understand the functional mechanisms of U...
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| Publicado no: | Exp Cell Res |
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| Main Authors: | , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2008
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6719340/ https://ncbi.nlm.nih.gov/pubmed/18561914 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yexcr.2008.05.001 |
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