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Gut IgA abundance in adult life is a major determinant of resistance to dextran sodium sulfate‐colitis and can compensate for the effects of inadequate maternal IgA received by neonates

Studies with gene‐deficient and gnotobiotic mice have identified many host and microbial factors that contribute to induced colitis, but information on whether specific factors determine susceptibility under more physiological conditions is lacking. Using wild‐type strains that differ in their IgA r...

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Dettagli Bibliografici
Pubblicato in:Immunology
Autori principali: Gupta, Suman, Basu, Srijani, Bal, Vineeta, Rath, Satyajit, George, Anna
Natura: Artigo
Lingua:Inglês
Pubblicazione: John Wiley and Sons Inc. 2019
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6700465/
https://ncbi.nlm.nih.gov/pubmed/31215020
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/imm.13091
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