Biphasic Impact of Prenatal Inflammation and Macrophage Depletion on the Wiring of Neocortical Inhibitory Circuits

The etiology of neurodevelopmental disorders is linked to defects in parvalbumin (PV)-expressing cortical interneurons and to prenatal immune challenges. Mouse models of maternal immune activation (MIA) and microglia deficits increase the postnatal density of PV interneurons, raising the question of...

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Detalhes bibliográficos
Publicado no:Cell Rep
Main Authors: Thion, Morgane Sonia, Mosser, Coralie-Anne, Férézou, Isabelle, Grisel, Pauline, Baptista, Sofia, Low, Donovan, Ginhoux, Florent, Garel, Sonia, Audinat, Etienne
Formato: Artigo
Idioma:Inglês
Publicado em: Cell Press 2019
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6685496/
https://ncbi.nlm.nih.gov/pubmed/31365857
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.celrep.2019.06.086
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