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Spine Ca(2+) Signaling in Spike-Timing-Dependent Plasticity
Calcium is a second messenger, which can trigger the modification of synaptic efficacy. We investigated the question of whether a differential rise in postsynaptic Ca(2+) ([Ca(2+)](i)) alone is sufficient to account for the induction of long-term potentiation (LTP) and long-term depression (LTD) of...
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| Gepubliceerd in: | J Neurosci |
|---|---|
| Hoofdauteurs: | , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
Society for Neuroscience
2006
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6674669/ https://ncbi.nlm.nih.gov/pubmed/17065442 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.1749-06.2006 |
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