Genetic and Physiological Evidence That Oligodendrocyte Gap Junctions Contribute to Spatial Buffering of Potassium Released during Neuronal Activity

Mice lacking the K(+) channel Kir4.1 or both connexin32 (Cx32) and Cx47 exhibit myelin-associated vacuoles, raising the possibility that oligodendrocytes, and the connexins they express, contribute to recycling the K(+) evolved during neuronal activity. To study this possibility, we first examined t...

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Detalhes bibliográficos
Publicado no:J Neurosci
Main Authors: Menichella, Daniela M., Majdan, Marta, Awatramani, Rajeshwar, Goodenough, Daniel A., Sirkowski, Erich, Scherer, Steven S., Paul, David L.
Formato: Artigo
Idioma:Inglês
Publicado em: Society for Neuroscience 2006
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Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6674647/
https://ncbi.nlm.nih.gov/pubmed/17065440
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.0304-06.2006
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