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Reversal of Brain Injury-Induced Prefrontal Glutamic Acid Decarboxylase Expression and Working Memory Deficits by D(1) Receptor Antagonism

Working memory (WM), the ability to transiently hold information in mind, is essential for high-level cognitive functions that are often impaired in brain-injured patients. The cellular and molecular mechanisms contributing to WM deficits, which can manifest in the absence of overt damage, in these...

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Detalles Bibliográficos
Publicado en:J Neurosci
Autores principales: Kobori, Nobuhide, Dash, Pramod K.
Formato: Artigo
Lenguaje:Inglês
Publicado: Society for Neuroscience 2006
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC6673989/
https://ncbi.nlm.nih.gov/pubmed/16624944
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.4687-05.2006
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