Accelerated Aβ Deposition in APPswe/PS1ΔE9 Mice with Hemizygous Deletions of TTR (Transthyretin)

A cardinal pathological lesion of Alzheimer's disease (AD) is the deposition of amyloid β (Aβ) in the brain. We previously reported that exposing transgenic mice harboring APPswe/PS1ΔE9 transgenes to an enriched environment resulted in reduced levels of Aβ peptides and deposition, findings that...

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Vydáno v:J Neurosci
Hlavní autoři: Choi, Se Hoon, Leight, Susan N., Lee, Virginia M.-Y., Li, Tong, Wong, Philip C., Johnson, Jeffrey A., Saraiva, Maria J., Sisodia, Sangram S.
Médium: Artigo
Jazyk:Inglês
Vydáno: Society for Neuroscience 2007
Témata:
Brief Communications
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC6672232/
https://ncbi.nlm.nih.gov/pubmed/17596449
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.1919-07.2007
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