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Ca(2+)/CaM Controls Ca(2+)-Dependent Inactivation of NMDA Receptors by Dimerizing the NR1 C Termini
Ca(2+) influx through NMDA receptors (NMDARs) leads to channel inactivation, which limits Ca(2+) entry and protects against excitotoxicity. Extensive functional data suggests that this Ca(2+)-dependent inactivation (CDI) requires both calmodulin (CaM) binding to the C0 cassette of the NR1 subunit�...
Kaydedildi:
| Yayımlandı: | J Neurosci |
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| Asıl Yazarlar: | , , , |
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
Society for Neuroscience
2008
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6671448/ https://ncbi.nlm.nih.gov/pubmed/18287503 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.5417-07.2008 |
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