Reciprocal activation within a kinase-effector complex underlying persistence of structural LTP

Long-term synaptic plasticity requires a mechanism that converts short Ca(2+) pulses into persistent biochemical signaling to maintain the changes in the synaptic structure and function. Here, we present a novel mechanism of a positive feedback loop, formed by a “reciprocally activating kinase-effec...

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Detalhes bibliográficos
Publicado no:Neuron
Main Authors: Saneyoshi, Takeo, Matsuno, Hitomi, Suzuki, Akio, Murakoshi, Hideji, Hedrick, Nathan G., Agnello, Emily, O’Connell, Rory, Stratton, Margaret M., Yasuda, Ryohei, Hayashi, Yasunori
Formato: Artigo
Idioma:Inglês
Publicado em: 2019
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6669903/
https://ncbi.nlm.nih.gov/pubmed/31078368
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuron.2019.04.012
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