Adenosine A(2A) Receptor Blockade Prevents Synaptotoxicity and Memory Dysfunction Caused by β-Amyloid Peptides via p38 Mitogen-Activated Protein Kinase Pathway

Alzheimer's disease (AD) is characterized by memory impairment, neurochemically by accumulation of β-amyloid peptide (namely Aβ(1-42)) and morphologically by an initial loss of nerve terminals. Caffeine consumption prevents memory dysfunction in different models, which is mimicked by antagonist...

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Veröffentlicht in:J Neurosci
Hauptverfasser: Canas, Paula M., Porciúncula, Lisiane O., Cunha, Geanne M. A., Silva, Carla G., Machado, Nuno J., Oliveira, Jorge M. A., Oliveira, Catarina R., Cunha, Rodrigo A.
Format: Artigo
Sprache:Inglês
Veröffentlicht: Society for Neuroscience 2009
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Articles
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC6665997/
https://ncbi.nlm.nih.gov/pubmed/19940169
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.3728-09.2009
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