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A Model for Sealing Plasmalemmal Damage in Neurons and Other Eukaryotic Cells
Plasmalemmal repair is necessary for survival of damaged eukaryotic cells. Ca(2+) influx through plasmalemmal disruptions activates calpain, vesicle accumulation at lesion sites, and membrane fusion proteins; Ca(2+) influx also initiates competing apoptotic pathways. Using the formation of a dye bar...
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| Publicado no: | J Neurosci |
|---|---|
| Main Authors: | , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Society for Neuroscience
2010
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6633751/ https://ncbi.nlm.nih.gov/pubmed/21106818 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.4155-10.2010 |
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