Erratum. hZnT8 (Slc30a8) Transgenic Mice That Overexpress the R325W Polymorph Have Reduced Islet Zn(2+) and Proinsulin Levels, Increased Glucose Tolerance After a High-Fat Diet, and Altered Levels of Pancreatic Zinc Binding Proteins. Diabetes 2017;66:551–559

Benzer Materyaller

• hZnT8 (Slc30a8) Transgenic Mice That Overexpress the R325W Polymorph Have Reduced Islet Zn(2+) and Proinsulin Levels, Increased Glucose Tolerance After a High-Fat Diet, and Altered Levels of Pancreatic Zinc Binding Proteins
  Yazar:: Li, Li, ve diğerleri
  Baskı/Yayın Bilgisi: (2017)
• C-Terminal Domain of the Human Zinc Transporter hZnT8 Is Structurally Indistinguishable from Its Disease Risk Variant (R325W)
  Yazar:: Ullah, Raheem, ve diğerleri
  Baskı/Yayın Bilgisi: (2020)
• Association of the SLC30A8 missense polymorphism R325W with proinsulin levels at baseline and after lifestyle, metformin or troglitazone intervention in the Diabetes Prevention Program
  Yazar:: Majithia, A. R., ve diğerleri
  Baskı/Yayın Bilgisi: (2011)
• Recognition of ZnT8, Proinsulin, and Homologous MAP Peptides in Sardinian Children at Risk of T1D Precedes Detection of Classical Islet Antibodies
  Yazar:: Niegowska, Magdalena, ve diğerleri
  Baskı/Yayın Bilgisi: (2016)
• Erratum. Direct Substrate Delivery Into Mitochondrial Fission–Deficient Pancreatic Islets Rescues Insulin Secretion. Diabetes 2017;66:1247–1257
  Yazar:: Kabra, Uma D., ve diğerleri
  Baskı/Yayın Bilgisi: (2017)