NK1.1(+) cells promote sustained tissue injury and inflammation after trauma with hemorrhagic shock

Various cell populations expressing NK1.1 contribute to innate host defense and systemic inflammatory responses, but their role in hemorrhagic shock and trauma remains uncertain. NK1.1(+) cells were depleted by i.p. administration of anti‐NK1.1 (or isotype control) on two consecutive days, followed...

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Detaylı Bibliyografya
Yayımlandı:J Leukoc Biol
Asıl Yazarlar: Chen, Shuhua, Hoffman, Rosemary A., Scott, Melanie, Manson, Joanna, Loughran, Patricia, Ramadan, Mostafa, Demetris, Anthony J., Billiar, Timothy R.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: John Wiley and Sons Inc. 2017
Konular:
Inflammation, Extracellular Mediators, & Effector Molecules
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6608053/
https://ncbi.nlm.nih.gov/pubmed/28515228
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1189/jlb.3A0716-333R
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