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Disruption of Foxg1 impairs neural plasticity leading to social and cognitive behavioral defects
The transcription factor Foxg1 is known to be continuously expressed at a high level in mature neurons in the telencephalon, but little is known about its role in neural plasticity. Mutations in human FOXG1 cause deficiencies in learning and memory and limit social ability, which is defined as FOXG1...
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| 出版年: | Mol Brain |
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| 主要な著者: | , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
BioMed Central
2019
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6599246/ https://ncbi.nlm.nih.gov/pubmed/31253171 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13041-019-0484-x |
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