Gas6 Attenuates Sepsis-Induced Tight Junction Injury and Vascular Endothelial Hyperpermeability via the Axl/NF-κB Signaling Pathway

Vascular endothelial functional dysregulation and barrier disruption are involved the initiation and development of sepsis. Growth arrest-specific protein 6 (Gas6), one of the endogenous ligands of TAM receptors (Tyro3, Axl, and Mertk), is confirmed to have beneficial functions in hemostasis, inflam...

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Publicat a:Front Pharmacol
Autors principals: Ni, Jingjing, Lin, Miaotong, Jin, Yangjie, Li, Jiajia, Guo, Yayong, Zhou, Jindong, Hong, Guangliang, Zhao, Guangju, Lu, Zhongqiu
Format: Artigo
Idioma:Inglês
Publicat: Frontiers Media S.A. 2019
Matèries:
Pharmacology
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC6585310/
https://ncbi.nlm.nih.gov/pubmed/31263416
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fphar.2019.00662
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