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Sodium nitroprusside evokes the release of immunoreactive calcitonin gene-related peptide and substance P from dorsal horn slices via nitric oxide-dependent and nitric oxide-independent mechanisms

The results of behavioral studies suggest that nitric oxide (NO) participates in certain spinal mechanisms that contribute to hyperalgesia. Additionally, previous studies indicate that the release of immunoreactive calcitonin gene-related peptide (iCGRP) and substance P (iSP) is increased in the dor...

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Detalhes bibliográficos
Publicado no:J Neurosci
Main Authors: Garry, MG, Richardson, JD, Hargreaves, KM
Formato: Artigo
Idioma:Inglês
Publicado em: Society for Neuroscience 1994
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6577028/
https://ncbi.nlm.nih.gov/pubmed/7517995
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.14-07-04329.1994
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