Sodium nitroprusside evokes the release of immunoreactive calcitonin gene-related peptide and substance P from dorsal horn slices via nitric oxide-dependent and nitric oxide-independent mechanisms

The results of behavioral studies suggest that nitric oxide (NO) participates in certain spinal mechanisms that contribute to hyperalgesia. Additionally, previous studies indicate that the release of immunoreactive calcitonin gene-related peptide (iCGRP) and substance P (iSP) is increased in the dor...

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Publicado en:J Neurosci
Autores principales: Garry, MG, Richardson, JD, Hargreaves, KM
Formato: Artigo
Lenguaje:Inglês
Publicado: Society for Neuroscience 1994
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Articles
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC6577028/
https://ncbi.nlm.nih.gov/pubmed/7517995
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.14-07-04329.1994
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