Neurochemical and histologic characterization of striatal excitotoxic lesions produced by the mitochondrial toxin 3-nitropropionic acid

An impairment of energy metabolism may underlie slow excitotoxic neuronal death in neurodegenerative diseases. We therefore examined the effects of intrastriatal, subacute systemic, or chronic systemic administration of the mitochondrial toxin 3-nitropropionic acid (3-NP) in rats. Following intrastr...

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Pubblicato in:J Neurosci
Autori principali: Beal, MF, Brouillet, E, Jenkins, BG, Ferrante, RJ, Kowall, NW, Miller, JM, Storey, E, Srivastava, R, Rosen, BR, Hyman, BT
Natura: Artigo
Lingua:Inglês
Pubblicazione: Society for Neuroscience 1993
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Articles
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6576392/
https://ncbi.nlm.nih.gov/pubmed/7692009
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.13-10-04181.1993
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