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Fetal and Maternal Hepcidin Respond Inversely to Prenatal Alcohol Exposure in a C57BL/6J Mouse Model (P09-010-19)
OBJECTIVES: Prenatal alcohol exposure (PAE) dysregulates iron metabolism and causes fetal iron deficiency in rats, even when mothers consume sufficient iron. This dysregulation is partly due to PAE-mediated induction of hepcidin, which routes iron to hepatic storage, where it is unavailable for feta...
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| Publicado no: | Curr Dev Nutr |
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| Main Authors: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Oxford University Press
2019
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6574878/ https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/cdn/nzz033.P09-010-19 |
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