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Dynorphin A-(1-17) induces alterations in free fatty acids, excitatory amino acids, and motor function through an opiate-receptor-mediated mechanism

The endogenous opioid dynorphin A-(1-17) (Dyn A) has been implicated as a mediator of tissue damage after traumatic spinal cord injury (TSCI) and causes hindlimb paralysis when administered intrathecally. Motor impairment following intrathecal Dyn A is attenuated by antagonists of excitatory amino a...

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Detalhes bibliográficos
Publicado no:J Neurosci
Main Authors: Bakshi, R, Newman, AH, Faden, AI
Formato: Artigo
Idioma:Inglês
Publicado em: Society for Neuroscience 1990
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6570042/
https://ncbi.nlm.nih.gov/pubmed/1980130
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.10-12-03793.1990
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