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Prostaglandins inhibit endogenous pain control mechanisms by blocking transmission at spinal noradrenergic synapses

Spinal intrathecal injections of the nonsteroidal antiinflammatory analgesics (NSAIAs) indomethacin and acetylsalicylic acid, which inhibit prostaglandin synthesis, cause dose-dependent hypoalgesia in the rat. Intrathecal injections of prostaglandin-E2 (PGE2) produce dose- dependent hyperalgesia. To...

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Publicat a:J Neurosci
Autors principals: Taiwo, YO, Levine, JD
Format: Artigo
Idioma:Inglês
Publicat: Society for Neuroscience 1988
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Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC6569250/
https://ncbi.nlm.nih.gov/pubmed/2833584
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.08-04-01346.1988
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