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Elevated synthesis of an axonally transported protein correlates with axon outgrowth in normal and injured pyramidal tracts
Axons of the adult mammalian CNS typically fail to regenerate after injury. Among the hypotheses to account for this failure is the proposition that certain axonal proteins necessary for axon growth are expressed in much greater abundance in developing than in mature neurons, and that these proteins...
Uloženo v:
| Vydáno v: | J Neurosci |
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| Hlavní autoři: | , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Society for Neuroscience
1986
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6568696/ https://ncbi.nlm.nih.gov/pubmed/3746423 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.06-09-02563.1986 |
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