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Elevated synthesis of an axonally transported protein correlates with axon outgrowth in normal and injured pyramidal tracts

Axons of the adult mammalian CNS typically fail to regenerate after injury. Among the hypotheses to account for this failure is the proposition that certain axonal proteins necessary for axon growth are expressed in much greater abundance in developing than in mature neurons, and that these proteins...

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Détails bibliographiques
Publié dans:J Neurosci
Auteurs principaux: Kalil, K, Skene, JH
Format: Artigo
Langue:Inglês
Publié: Society for Neuroscience 1986
Sujets:
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC6568696/
https://ncbi.nlm.nih.gov/pubmed/3746423
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.06-09-02563.1986
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