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Elevated synthesis of an axonally transported protein correlates with axon outgrowth in normal and injured pyramidal tracts

Axons of the adult mammalian CNS typically fail to regenerate after injury. Among the hypotheses to account for this failure is the proposition that certain axonal proteins necessary for axon growth are expressed in much greater abundance in developing than in mature neurons, and that these proteins...

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Detalhes bibliográficos
Publicado no:J Neurosci
Main Authors: Kalil, K, Skene, JH
Formato: Artigo
Idioma:Inglês
Publicado em: Society for Neuroscience 1986
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6568696/
https://ncbi.nlm.nih.gov/pubmed/3746423
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.06-09-02563.1986
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