Analysis of deletional hereditary persistence of fetal hemoglobin/δβ‐thalassemia and δ‐globin gene mutations in Southerwestern China

Lignende værker

• The prevalence and molecular characterization of (δβ)(0)‐thalassemia and hereditary persistence of fetal hemoglobin in the Chinese Zhuang population
  af: He, Sheng, et al.
  Udgivet: (2017)
• Molecular epidemiology and hematologic characterization of δβ-thalassemia and hereditary persistence of fetal hemoglobin in 125,661 families of greater Guangzhou area, the metropolis of southern China
  af: Jiang, Fan, et al.
  Udgivet: (2020)
• Featured Article: Modulation of fetal hemoglobin in hereditary persistence of fetal hemoglobin deletion type-2, compared to Sicilian δβ-thalassemia, by BCL11A and SOX6-targeting microRNAs
  af: Fornari, Thais A, et al.
  Udgivet: (2016)
• Total ginsenosides enhance γ-globin expression and fetal hemoglobin production in β-thalassemia models
  af: Dongling Cai, et al.
  Udgivet: (2025-08-01)
• Low fetal hemoglobin rates in patients carrying Thai (δβ)(0)-deletion and Turkish (δβ)(0)-deletion/inversion strengthen the hypothesis that the 5′δ BCL11A binding site plays a major role in its fetal hemoglobin inhibitory regulation. Response to “The 12.6 kb-deletion in the β-globin gene cluster is the known Thai/Vietnamese (δβ)(0)-thalassemia commonly found in Southeast Asia”
  af: Ghedira, Elyes Slim, et al.
  Udgivet: (2013)