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Elevated Kallikrein-binding protein in diabetes impairs wound healing through inducing macrophage M1 polarization
BACKGROUND: The accumulation of M1-polarized macrophages and excessive inflammation are important in the pathogenesis of diabetic foot ulcer (DFU). However, the underlying mechanism of DFU pathogenesis and the crucial regulators of DFU are less well known. Our previous study reported that kallikrein...
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| Vydáno v: | Cell Commun Signal |
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| Hlavní autoři: | , , , , , , , , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
BioMed Central
2019
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6558923/ https://ncbi.nlm.nih.gov/pubmed/31182110 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12964-019-0376-9 |
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