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SAT-453 Diabetic "Ketoalkalosis" Due to SGLT2 Inhibitor in the Setting of Cushing's Disease
Introduction - The combination of sodium glucose cotransporter-2 inhibitor (SGLT-2i) use and glucocorticoid excess can trigger ketone production by reducing insulin secretion/sensitivity and stimulating glucosuria, glucagon production, and lipolysis. In addition, metabolic alkalosis caused by glucoc...
Tallennettuna:
| Julkaisussa: | J Endocr Soc |
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| Päätekijät: | , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
Endocrine Society
2019
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6552005/ https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1210/js.2019-SAT-453 |
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