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SAT-163 Prolactin Receptor Signaling Regulates a Pregnancy-Specific Transcriptional Program in Mouse Islets

Pancreatic β-cells undergo profound hyperplasia during pregnancy to maintain maternal euglycemia. Failure to reprogram β-cells into a more replicative state has been found to underlie susceptibility to gestational diabetes (GDM). Our laboratory has recently identified a requirement for prolactin rec...

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Detalhes bibliográficos
Publicado no:J Endocr Soc
Main Authors: Pepin, Mark, Wende, Adam, Banerjee, Ronadip
Formato: Artigo
Idioma:Inglês
Publicado em: Endocrine Society 2019
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6551691/
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1210/js.2019-SAT-163
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