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Chronic synaptic insulin resistance after traumatic brain injury abolishes insulin protection from amyloid beta and tau oligomer-induced synaptic dysfunction

Traumatic brain injury (TBI) is a risk factor for Alzheimer’s disease (AD), although the mechanisms contributing to this increased risk are unknown. Insulin resistance is an additional risk factor for AD whereby decreased insulin signaling increases synaptic sensitivity to amyloid beta (Aβ) and tau....

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Detaylı Bibliyografya
Yayımlandı:Sci Rep
Asıl Yazarlar: Franklin, Whitney, Krishnan, Balaji, Taglialatela, Giulio
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Nature Publishing Group UK 2019
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6546708/
https://ncbi.nlm.nih.gov/pubmed/31160730
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-019-44635-z
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