Circulating miR-103a-3p contributes to angiotensin II-induced renal inflammation and fibrosis via a SNRK/NF-κB/p65 regulatory axis

Although angiotensin II (AngII) is known to cause renal injury and fibrosis, the underlying mechanisms remain poorly characterized. Here we show that hypertensive nephropathy (HN) patients and AngII-infused mice exhibit elevated levels of circulating miR103a-3p. We observe a positive correlation bet...

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Detalhes bibliográficos
Publicado no:Nat Commun
Main Authors: Lu, Qiulun, Ma, Zejun, Ding, Ye, Bedarida, Tatiana, Chen, Liming, Xie, Zhonglin, Song, Ping, Zou, Ming-Hui
Formato: Artigo
Idioma:Inglês
Publicado em: Nature Publishing Group UK 2019
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6513984/
https://ncbi.nlm.nih.gov/pubmed/31086184
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41467-019-10116-0
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