Ionic silicon improves endothelial cells’ survival under toxic oxidative stress by overexpressing angiogenic markers and antioxidant enzymes

Oxidative stress, induced by harmful levels of reactive oxygen species, is a common occurrence that impairs proper bone defect vascular healing through the impairment of endothelial cell function. Ionic silicon released from silica-based biomaterials, can upregulate hypoxia-inducible factor-1α (HIF-...

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Detaylı Bibliyografya
Yayımlandı:J Tissue Eng Regen Med
Asıl Yazarlar: Monte, Felipe, Cebe, Tugba, Ripperger, Daniel, Ighani, Fareed, Kojouharov, Hristo V., Chen, Benito M., Kim, Harry K. W., Aswath, Pranesh B., Varanasi, Venu G.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2018
Konular:
Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6508967/
https://ncbi.nlm.nih.gov/pubmed/30062712
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/term.2744
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