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HC diet inhibited testosterone synthesis by activating endoplasmic reticulum stress in testicular Leydig cells
Emerging epidemiological studies indicate that hypercholesterolaemia is a risk factor for testosterone deficiency. However, the underlying mechanism is unclear. Testicular Leydig cells are the primary source of testosterone in males. To identify the effect and mechanism of cholesterol overload on Le...
Uloženo v:
| Vydáno v: | J Cell Mol Med |
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| Hlavní autoři: | , , , , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
John Wiley and Sons Inc.
2019
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6484377/ https://ncbi.nlm.nih.gov/pubmed/30884106 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.14143 |
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