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Nuclear MET requires ARF and is inhibited by carbon nanodots through binding to phosphor-Tyrosine in prostate cancer

Nuclear receptor tyrosine kinases (nRTKs) are aberrantly upregulated in many types of cancers, but the regulation of nRTK remains unclear. We previously showed androgen deprivation therapy (ADT) induces nMET in castration resistant prostate cancer (CRPC) specimens. Through gene expression microarray...

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Dettagli Bibliografici
Pubblicato in:Oncogene
Autori principali: Xie, Yingqiu, Fan, Haiyan, Lu, Wenfu, Yang, Qing, Nurkesh, Ayan, Yeleusizov, Tleubek, Maipas, Aisulu, Lu, Jiang, Manarbek, Limara, Chen, Zhenbang, Benassi, Enrico
Natura: Artigo
Lingua:Inglês
Pubblicazione: 2018
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6474787/
https://ncbi.nlm.nih.gov/pubmed/30568225
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41388-018-0608-2
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