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Nuclear MET requires ARF and is inhibited by carbon nanodots through binding to phosphor-Tyrosine in prostate cancer
Nuclear receptor tyrosine kinases (nRTKs) are aberrantly upregulated in many types of cancers, but the regulation of nRTK remains unclear. We previously showed androgen deprivation therapy (ADT) induces nMET in castration resistant prostate cancer (CRPC) specimens. Through gene expression microarray...
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| Pubblicato in: | Oncogene |
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| Autori principali: | , , , , , , , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
2018
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6474787/ https://ncbi.nlm.nih.gov/pubmed/30568225 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41388-018-0608-2 |
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