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TLR4 participates in sympathetic hyperactivity Post-MI in the PVN by regulating NF-κB pathway and ROS production

Sympathetic nerve hyperactivity is a primary reason for fatal ventricular arrhythmias (VAs) following myocardial infarction (MI). Pro-inflammatory cytokines produced in the paraventricular nucleus (PVN) post-MI are associated with sympathetic overexcitation; however, the precise mechanism needs furt...

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Kaydedildi:
Detaylı Bibliyografya
Yayımlandı:Redox Biol
Asıl Yazarlar: Wang, Yu, Hu, Hesheng, Yin, Jie, Shi, Yugen, Tan, Jiayu, Zheng, Lu, Wang, Cailing, Li, Xiaolu, Xue, Mei, Liu, Ju, Wang, Ye, Li, Yan, Li, Xinran, Liu, Fuhong, Liu, Qiang, Yan, Suhua
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Elsevier 2019
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6460304/
https://ncbi.nlm.nih.gov/pubmed/30978539
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.redox.2019.101186
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