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Oxidative damage of SP-D abolishes control of eosinophil extracellular DNA trap formation

The asthmatic airways are highly susceptible to inflammatory injury by air pollutants such as ozone (O(3)), characterized by enhanced activation of eosinophilic granulocytes and a failure of immune protective mechanisms. Eosinophil activation during asthma exacerbation contributes to the proinflamma...

Täydet tiedot

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Bibliografiset tiedot
Julkaisussa:J Leukoc Biol
Päätekijät: Yousefi, Shida, Sharma, Satish K., Stojkov, Darko, Germic, Nina, Aeschlimann, Salome, Ge, Moyar Q., Flayer, Cameron H., Larson, Erik D., Redai, Imre G., Zhang, Suhong, Koziol-White, Cynthia J., Karikó, Katalin, Simon, Hans-Uwe, Haczku, Angela
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 2018
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC6450398/
https://ncbi.nlm.nih.gov/pubmed/29733456
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/JLB.3AB1117-455R
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