Blocking VEGF by Bevacizumab Compromises Electrophysiological and Morphological Properties of Hippocampal Neurons

A hallmark of glioblastoma multiforme (GBM) is neoangiogenesis, mediated by the overexpression of vascular endothelial growth factor (VEGF). Anti-VEGF antibodies, like bevacizumab, prolong progression-free survival in GBM, however, this treatment has been reported to be associated with a decline in...

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Detalhes bibliográficos
Publicado no:Front Cell Neurosci
Main Authors: Latzer, Pauline, Shchyglo, Olena, Hartl, Tim, Matschke, Veronika, Schlegel, Uwe, Manahan-Vaughan, Denise, Theiss, Carsten
Formato: Artigo
Idioma:Inglês
Publicado em: Frontiers Media S.A. 2019
Assuntos:
Neuroscience
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6445260/
https://ncbi.nlm.nih.gov/pubmed/30971896
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fncel.2019.00113
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