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Palmitic acid-induced autophagy increases reactive oxygen species via the Ca(2+)/PKCα/NOX4 pathway and impairs endothelial function in human umbilical vein endothelial cells
It is well known that the lipotoxic mechanism of palmitic acid (PA), a main constituent of triglyceride, is dependent on reactive oxygen species (ROS). Recently, it has also been reported that PA is an autophagy inducer. However, the causal association and underlying mechanism of induced autophagy a...
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| 出版年: | Exp Ther Med |
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| 主要な著者: | , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
D.A. Spandidos
2019
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6425131/ https://ncbi.nlm.nih.gov/pubmed/30906429 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3892/etm.2019.7269 |
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