Coincident diabetes mellitus modulates Th1-, Th2-, and Th17-cell responses in latent tuberculosis in an IL-10- and TGF-β-dependent manner

Type 2 diabetes mellitus (DM) is a risk factor for the development of active tuberculosis (TB), although its role in the TB-induced responses in latent TB (LTB) is not well understood. Since Th1, Th2, and Th17 responses are important in immunity to LTB, we postulated that coincident DM could alter t...

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Detaylı Bibliyografya
Yayımlandı:Eur J Immunol
Asıl Yazarlar: Kumar, Nathella Pavan, Moideen, Kadar, George, Parakkal Jovvian, Dolla, Chandrakumar, Kumaran, Paul, Babu, Subash
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2015
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Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6340057/
https://ncbi.nlm.nih.gov/pubmed/26518995
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/eji.201545973
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