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Oxytocin can regulate myometrial smooth muscle excitability by inhibiting the Na(+)‐activated K(+) channel, Slo2.1
KEY POINTS: At the end of pregnancy, the uterus transitions from a quiescent state to a highly contractile state. This transition requires that the uterine (myometrial) smooth muscle cells increase their excitability, although how this occurs is not fully understood. We identified SLO2.1, a potassiu...
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| 出版年: | J Physiol |
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| 主要な著者: | , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
John Wiley and Sons Inc.
2018
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6312452/ https://ncbi.nlm.nih.gov/pubmed/30334255 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/JP276806 |
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