Exchange protein activated by cyclic-adenosine monophosphate (Epac) regulates atrial fibroblast function and controls cardiac remodelling

AIMS: Heart failure (HF) produces left atrial (LA)-selective fibrosis and promotes atrial fibrillation. HF also causes adrenergic activation, which contributes to remodelling via a variety of signalling molecules, including the exchange protein activated by cAMP (Epac). Here, we evaluate the effects...

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Vydáno v:Cardiovasc Res
Hlavní autoři: Surinkaew, Sirirat, Aflaki, Mona, Takawale, Abhijit, Chen, Yu, Qi, Xiao-Yan, Gillis, Marc-Antoine, Shi, Yan-Fen, Tardif, Jean-Claude, Chattipakorn, Nipon, Nattel, Stanley
Médium: Artigo
Jazyk:Inglês
Vydáno: Oxford University Press 2019
Témata:
Original Articles
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC6302269/
https://ncbi.nlm.nih.gov/pubmed/30016400
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/cvr/cvy173
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