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An expanding network of cytoskeletal defects
In this issue of Blood, Brigida et al(1) demonstrate that null mutations in ARPC1B result in combined immunodeficiency because of defects in T-cell migration, lymphoproliferation, and formation of the immune synapse, and further show that these abnormalities may be rescued by transduction of wild-ty...
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| Publicado no: | Blood |
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| Autor principal: | |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Society of Hematology
2018
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6265650/ https://ncbi.nlm.nih.gov/pubmed/30498066 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2018-10-878603 |
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