Complement 1q-like-3 protein inhibits insulin secretion from pancreatic β-cells via the cell adhesion G protein–coupled receptor BAI3

Secreted proteins are important metabolic regulators in both healthy and disease states. Here, we sought to investigate the mechanism by which the secreted protein complement 1q-like-3 (C1ql3) regulates insulin secretion from pancreatic β-cells, a key process affecting whole-body glucose metabolism....

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Gepubliceerd in:J Biol Chem
Hoofdauteurs: Gupta, Rajesh, Nguyen, Dan C., Schaid, Michael D., Lei, Xia, Balamurugan, Appakalai N., Wong, G. William, Kim, Jeong-a, Koltes, James E., Kimple, Michelle E., Bhatnagar, Sushant
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: American Society for Biochemistry and Molecular Biology 2018
Onderwerpen:
Metabolism
Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC6254345/
https://ncbi.nlm.nih.gov/pubmed/30228187
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.RA118.005403
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