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Hepatitis B virus promotes β-catenin-signalling and disassembly of adherens junctions in a Src kinase dependent fashion
Hepatitis B virus (HBV) infection is a prominent cause of hepatocellular carcinoma (HCC) but the underlying molecular mechanisms are complex and multiple pathways have been proposed such as the activation of the Wnt−/β-catenin-signalling and dysregulation of E-cadherin/β-catenin adherens junctions....
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| Vydáno v: | Oncotarget |
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| Hlavní autoři: | , , , , , , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Impact Journals LLC
2018
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6188061/ https://ncbi.nlm.nih.gov/pubmed/30338037 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/oncotarget.26103 |
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