Promyelocytic leukemia (PML) nuclear bodies (NBs) induce latent/quiescent HSV-1 genomes chromatinization through a PML NB/Histone H3.3/H3.3 Chaperone Axis

Herpes simplex virus 1 (HSV-1) latency establishment is tightly controlled by promyelocytic leukemia (PML) nuclear bodies (NBs) (or ND10), although their exact contribution is still elusive. A hallmark of HSV-1 latency is the interaction between latent viral genomes and PML NBs, leading to the forma...

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Dettagli Bibliografici
Pubblicato in:PLoS Pathog
Autori principali: Cohen, Camille, Corpet, Armelle, Roubille, Simon, Maroui, Mohamed Ali, Poccardi, Nolwenn, Rousseau, Antoine, Kleijwegt, Constance, Binda, Olivier, Texier, Pascale, Sawtell, Nancy, Labetoulle, Marc, Lomonte, Patrick
Natura: Artigo
Lingua:Inglês
Pubblicazione: Public Library of Science 2018
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Research Article
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6168178/
https://ncbi.nlm.nih.gov/pubmed/30235352
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.ppat.1007313
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