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Inhibiting neutral amino acid transport for the treatment of phenylketonuria

The neuropathological effects of phenylketonuria (PKU) stem from the inability of the body to metabolize excess phenylalanine (Phe), resulting in accumulation of Phe in the blood and brain. Since the kidney normally reabsorbs circulating amino acids with high efficiency, we hypothesized that prevent...

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Detalhes bibliográficos
Publicado no:JCI Insight
Main Authors: Belanger, Adam M., Przybylska, Malgorzata, Gefteas, Estelle, Furgerson, Matthew, Geller, Sarah, Kloss, Alla, Cheng, Seng H., Zhu, Yunxiang, Yew, Nelson S.
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Clinical Investigation 2018
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6124451/
https://ncbi.nlm.nih.gov/pubmed/30046012
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/jci.insight.121762
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