Paradoxical counteraction by imatinib against cell death in myeloid progenitor 32D cells expressing p210BCR-ABL

Chronic myeloid leukemia (CML) is believed to be caused by the tyrosine kinase p210BCR-ABL, which exhibits growth-promoting and anti-apoptotic activities. However, mechanisms that allow cell differentiation in CML still remain elusive. Here we established tetracycline (Tet)-regulatable p210BCR-ABL-e...

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Dades bibliogràfiques
Publicat a:Oncotarget
Autors principals: Takita, Morichika, Tsukahara, Fujiko, Mishima, Taishi, Ieguchi, Katsuaki, Yamada, Masayuki, Honda, Hiroaki, Maru, Yoshiro
Format: Artigo
Idioma:Inglês
Publicat: Impact Journals LLC 2018
Matèries:
Research Paper
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC6114964/
https://ncbi.nlm.nih.gov/pubmed/30167087
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/oncotarget.25849
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