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Tumor cross-talk networks promote growth and support immune evasion in pancreatic cancer
In the event of an injury, normal tissues exit quiescent homeostasis and rapidly engage a complex stromal and immune program. These tissue repair responses are hijacked and become dysregulated in carcinogenesis to form a growth-supportive tumor microenvironment. In pancreatic ductal adenocarcinoma (...
Αποθηκεύτηκε σε:
| Τόπος έκδοσης: | Am J Physiol Gastrointest Liver Physiol |
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| Κύριοι συγγραφείς: | , , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
American Physiological Society
2018
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6109710/ https://ncbi.nlm.nih.gov/pubmed/29543507 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpgi.00416.2017 |
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