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Tumor cross-talk networks promote growth and support immune evasion in pancreatic cancer

In the event of an injury, normal tissues exit quiescent homeostasis and rapidly engage a complex stromal and immune program. These tissue repair responses are hijacked and become dysregulated in carcinogenesis to form a growth-supportive tumor microenvironment. In pancreatic ductal adenocarcinoma (...

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Bibliografske podrobnosti
izdano v:Am J Physiol Gastrointest Liver Physiol
Main Authors: Halbrook, Christopher J., Pasca di Magliano, Marina, Lyssiotis, Costas A.
Format: Artigo
Jezik:Inglês
Izdano: American Physiological Society 2018
Teme:
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC6109710/
https://ncbi.nlm.nih.gov/pubmed/29543507
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpgi.00416.2017
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