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Tumor cross-talk networks promote growth and support immune evasion in pancreatic cancer
In the event of an injury, normal tissues exit quiescent homeostasis and rapidly engage a complex stromal and immune program. These tissue repair responses are hijacked and become dysregulated in carcinogenesis to form a growth-supportive tumor microenvironment. In pancreatic ductal adenocarcinoma (...
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| Gepubliceerd in: | Am J Physiol Gastrointest Liver Physiol |
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| Hoofdauteurs: | , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
American Physiological Society
2018
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6109710/ https://ncbi.nlm.nih.gov/pubmed/29543507 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpgi.00416.2017 |
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