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Salbutamol‐induced electrophysiological changes show no correlation with electrophysiological changes during hyperinsulinaemic–hypoglycaemic clamp in young people with Type 1 diabetes

AIMS: Hypoglycaemia causes QT‐interval prolongation and appears pro‐arrhythmogenic. Salbutamol, a β(2)‐adrenoreceptor agonist also causes QT‐interval prolongation. We hypothesized that the magnitude of electrophysiological changes induced by salbutamol and hypoglycaemia might relate to each other an...

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Detaylı Bibliyografya
Yayımlandı:Diabet Med
Asıl Yazarlar: Novodvorsky, P., Bernjak, A., Robinson, E. J., Iqbal, A., Macdonald, I. A., Jacques, R. M., Marques, J. L. B., Sheridan, P. J., Heller, S. R.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: John Wiley and Sons Inc. 2018
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6099209/
https://ncbi.nlm.nih.gov/pubmed/29682793
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/dme.13650
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