Helicobacter pylori infection downregulates duodenal CFTR and SLC26A6 expressions through TGFβ signaling pathway

BACKGROUND: The pathogenesis of Helicobacter pylori (H. pylori) infection-induced duodenal ulcer remains to be elucidated. Duodenal mucosal bicarbonate secretion is the most important protective factor against acid-induced mucosal injury. We previously revealed that H. pylori infection downregulated...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:BMC Microbiol
Päätekijät: Wen, Guorong, Deng, Shili, Song, Wenfeng, Jin, Hai, Xu, Jingyu, Liu, Xuemei, Xie, Rui, Song, Penghong, Tuo, Biguang
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: BioMed Central 2018
Aiheet:
Research Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC6098588/
https://ncbi.nlm.nih.gov/pubmed/30119655
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12866-018-1230-8
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