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Altered Mitochondrial Acetylation Profiles in a Kainic Acid Model of Temporal Lobe Epilepsy

Impaired bioenergetics and oxidative damage in the mitochondria are implicated in the etiology of temporal lobe epilepsy, and hyperacetylation of mitochondrial proteins has recently emerged as a critical negative regulator of mitochondrial functions. However, the roles of mitochondrial acetylation a...

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Dettagli Bibliografici
Pubblicato in:Free Radic Biol Med
Autori principali: Gano, Lindsey B., Liang, Li-Ping, Ryan, Kristen, Michel, Cole R., Gomez, Joe, Vassilopoulos, Athanassios, Reisdorph, Nichole, Fritz, Kristofer S., Patel, Manisha
Natura: Artigo
Lingua:Inglês
Pubblicazione: 2018
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC6082368/
https://ncbi.nlm.nih.gov/pubmed/29778462
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.freeradbiomed.2018.05.063
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